J s cheigh (10 Ergebnisse)
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Taschenbuch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a 'young' disorder whose existence has been known for less than a century. It… is not only extremely prevalent among every popula tion, but also deleterious to the health of mankind. The more we understand about hypertension's harmful effects, the more urgent is the need for its effective control. The kidney is the central organ that controls vascular tone and body fluid volume; these two factors are dominant in determining arterial blood pres sure. Hence, it is not surprising to find in hypertensive disorders that there are abnormalities in the kidneys, functional or anatomical, subtle or overt, that cause or are the consequence of hypertension. The first suggestion that the kidney could cause hypertension was made in 1836, before arterial pressure could even be measured, by Richard Bright. He observed that cardiac hypertrophy was often present in patients who died of renal disease. It was, however, Goldblatt and his colleagues in 1934 who opened the modern era of experimental and clinical research in renal hypertension. Since then, although far from complete, enthusiastic and intensive research efforts have greatly improved our understanding of the nature of renal hypertension.
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Taschenbuch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - 1. Evaluation of patients with renal disease.- 1. Introduction.- 2. Identifying Renal Disease.- 2.1. History.- 2.2. Family history.- 2.3. Physical examination.- 2.4. Urinalysis.- 2.5. The chemical data base.- 2.6. Radiologic techniques.- 2.7. Percuta…neous renal biopsy.- 2.8. Risks and cost of procedures.- 3. Syndromes of Renal Disease.- 3.1. Acute renal failure.- 3.2. Chronic renal failure.- 3.3. Acute nephritic syndrome.- 3.4. Nephrotic syndrome.- 3.5. Urinary tract infection.- 3.6. Obstructive nephropathy.- 3.7. Renal tubular dysfunction.- 3.8. Nephrolithiasis.- 3.9. Accelerated hypertension.- 4. Conclusion.- 2. Disorders of water, sodium and potassium metabolism.- 1. Maintenance of Osmotic Homeostasis.- 1.1. General considerations.- 1.2. Free-water clearance.- 1.3. Renal concentrating and diluting mechanisms.- 1.4. Antidiuretic hormone.- 1.5. Summary of requirements for maximum water diuresis $$left( {{C_{{H_2}O}}}ight)$$.- 2. Clinical States Associated with Hypo-osmolality.- 2.1. True dilutional hyponatremia.- 2.2. Renal failure.- 2.3. Decreased delivery of filtrate to diluting segment of the nephron.- 2.4. Syndrome of inappropriate ADH (SIADH).- 2.5 Drugs associated with impaired free-water clearance.- 2.6. Endocrine deficiencies.- 2.7. Reset osmostat.- 2.8. Summary of the diagnostic approach to hyponatremia.- 3. Hypernatremia and Disorders of Urine Concentration.- 3.1. Low circulating levels of ADH.- 3.2. Renal tubular hyporesponsiveness to ADH (nephrogenic diabetes insipidus).- 3.3. Diagnostic approach to the patient with a disorder of urinary concentration.- 4. Disorders of Sodium Metabolism.- 4.1. Determinants of renal sodium excretion.- 4.2. Sodium retaining states (edema).- 4.3. Diuretic therapy.- 5. Disorders of Potassium Metabolism.- 5.1. Internal potassium balance.- 5.2. External potassium balance.- 5.3. Hyperkalemia.- 5.4. Hypokalemia.- 3. Acid-base disturbances.- 1. Introduction.- 1.1. Physiologic buffers.- 1.2. Respiratory control of pH.- 1.3. Renal control of H + excretion.- 1.4. Clinical definitions.- 1.5. Diagnostic approach.- 2. Metabolic Acidosis.- 2.1. Anion gap.- 2.2. Metabolic acidosis with an increased anion gap.- 2.3. Metabolic acidosis associated with a normal anion gap (hyperchloremic).- 2.4. Treatment of metabolic acidosis.- 3. Metabolic Alkalosis.- 3.1. Metabolic alkalosis associated with low urinary Cl -.- 3.2. Metabolic alkalosis associated with normal or increased urinary Cl -.- 3.3. Treatment of metabolic alkalosis.- 4. Respiratory Alkalosis.- 5. Respiratory Acidosis.- 6. Mixed Acid-Base Disorders.- 4. Glomerulonephropathies.- 1. Introduction.- 2. Asymptomatic Urinary Abnormalities.- 2.1. Hematuria.- 2.2. Asymptomatic proteinuria.- 2.3. Asymptomatic proteinuria and hematuria.- 3. Nephrotic Syndrome.- 3.1. Definition.- 3.2. Pathogenesis of proteinuria.- 3.3. Patient presentation and evaluation.- 3.4. Clinicopathological correlation.- 4. Acute Nephritic Syndrome.- 4.1. Definition.- 4.2. Patient presentation and evaluation.- 4.3. Clinicopathological correlation.- 4.4. Management of acute nephritic syndrome.- 4.5. Prognosis.- 5. Rapidly Progressive Glomerulonephritis (RPGN).- 5.1. Definition.- 5.2. Patient presentation and evaluation.- 5.3. Clinicopathological correlation.- 6. Chronic Nephritic Syndrome.- 6.1. Definition.- 6.2. Patient presentation and evaluation.- 7. Glomerulonephritis and Systemic Disease.- 7.1. Metabolic and inherited diseases.- 7.2. Hereditary nephritis.- 7.3. Infectious disease.- 7.4. Toxic nephropathy.- 7.5. Collagen vascular disease.- 7.6. Dysproteinemias.- 7.7. Pregnancy.- 5. Urinary tract infection and pyelonephritis.- 1. Prevalence.- 1.1. Overall incidence.- 1.2. Prevalence of urinary tract infection in children.- 1.3. Prevalence of urinary tract infection in adults.- 2. Clinical Significance of Bacteriuria.- 2.1. Pregnancy and urinary tract infection.- 2.2. Hypertension and urinary tract infection.- 2.3. Diabetes and urinary tract infection.- 2.4. Bacteriuria in non-pregnant women.- 2.5. Bacteriuria in children.- 2.6. Bacteriuria in men.- 2.7. Nosocomial urinary tract infections.- 3. Pathogenesis.- 3.1. Local barriers of invasion.- 3.2. Cervico-vaginal antibodies.- 3.3. Uroepithelial antibodies.- 3.4. Systemic antibody response.- 3.5. Other mechanisms.- 3.6. Vesicoureteral valve.- 4. Etiology.- 4.1. Bacterial infection.- 4.2. Fungal infections.- 4.3. Viral infection.- 5. Clinical Manifestations.- 6. Diagnosis.- 6.1. Urine collection.- 6.2. Microscopic examination.- 6.3. Localization.- 7. Management.- 7.1. Asymptomatic bacteriuria.- 7.2. Acute pyelonephritis.- 7.3. Recurrent infections.- 7.4. Antibiotics.- 7.5. Prophylaxis.- 6. Tubulo-interstitial nephritis.- 1. Hereditary Kidney Diseases.- 1.1. Medullary cystic disease.- 1.2. Medullary sponge kidney.- 1.3. Hereditary familial nephritis (Alport¿s syndrome).- 2. Metabolic Kidney Diseases.- 2.1. Hypercalcemia.- 2.2. Hypokalemia.- 2.3. Oxalate nephropathy.- 2.4. Gouty nephropathy.- 3. Hematologic Diseases.- 3.1. Hemolytic-uremic syndrome.- 3.2. Sickle cell anemia.- 4. Vascular Diseases.- 5. Neoplastic Diseases.- 6. Infections.- 7. Immunological Diseases.- 7.1. Antitubular basement membrane antibodies.- 7.2. Immune complex interstitial nephritis.- 7.3. Reflux nephropathy.- 7.4. Drug-related interstitial nephritis.- 7.5. Renal allografts.- 7.6. Sj¿gren¿s syndrome.- 8. Analgesic Nephropathy.- 8.1. Aspirin.- 8.2. Phenacetin.- 8.3. Pathology.- 8.4. Pathogenesis.- 8.5. Clinical manifestations.- 9. Heavy Metals.- 10. Balkan Nephropathy.- 11. Radiation Nephritis.- 7. Cystic diseases of the kidney.- 1. Introduction.- 2. Cystic Dysplasia.- 2.1. Renal aplasia.- 2.2. Congenital multicystic kidney.- 2.3. Pathology.- 2.4. Etiology and pathogenesis.- 2.5. Clinical presentation.- 2.6. Diagnosis.- 2.7. Prognosis and therapy.- 3. Polycystic Disease.- 3.1. Infantile polycystic disease.- 3.2. Adult polycystic disease.- 4. Medullary Cysts of the Kidney.
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Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a 'young' disorder whose existence has been known for less than a century. It is not… only extremely prevalent among every popula tion, but also deleterious to the health of mankind. The more we understand about hypertension's harmful effects, the more urgent is the need for its effective control. The kidney is the central organ that controls vascular tone and body fluid volume; these two factors are dominant in determining arterial blood pres sure. Hence, it is not surprising to find in hypertensive disorders that there are abnormalities in the kidneys, functional or anatomical, subtle or overt, that cause or are the consequence of hypertension. The first suggestion that the kidney could cause hypertension was made in 1836, before arterial pressure could even be measured, by Richard Bright. He observed that cardiac hypertrophy was often present in patients who died of renal disease. It was, however, Goldblatt and his colleagues in 1934 who opened the modern era of experimental and clinical research in renal hypertension. Since then, although far from complete, enthusiastic and intensive research efforts have greatly improved our understanding of the nature of renal hypertension.
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Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - .an excellent introduction to clinical nephrology and, therefore, a very helpful text for medical students, house officers, and even beginning fellows in clinical nephrology.' Dialysis and Transplantation, 12: 1 (1983).
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Zustand: New. Editor(s): Cheigh, Jhoong S.; Stenzel, Kurt H.; Rubin, Albert L. Series: Developments in Nephrology. Num Pages: 312 pages, biography. BIC Classification: MJR. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly. Dimension: 235 x 155 x 19. Weight in Grams: 636. . 1986. Hardback. . . . .…Books ship from the US and Ireland.
