doevendans s a (13 Ergebnisse)

Zustand: Good. Vouwtjes/kreukjes kaft.

24 cm. original hardcover. xviii,276 pp. ills, diagrams. references. index. -(very) good. 655g.

Hardcover. Zustand: Good. No Jacket. Pages can have notes/highlighting. Spine may show signs of wear. ~ ThriftBooks: Read More, Spend Less.

Zustand: New. This accessible handbook aims to help decision-making in cardiovascular patients with a potentially inherited disease. Clinicians from around the globe discuss the relevance of genetics for their field of interest covering atherosclerosis, cardiomyopathy and arrhythmias. Editor(s): Doevendans, P. A. F. M.; Wilde, Arthur A. M. Series: Developments in Cardiovascular Medicine. Num Pages: 226 pages, biography. BIC Classification: MFN; MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly. Dimension: 235 x 155 x 16. Weight in Grams: 589. . 2001. Hardback. . . . . Books ship from the US and Ireland.

Zustand: New. In.

Zustand: New. In.

Taschenbuch. Zustand: Neu. Cardiovascular Specific Gene Expression | P. A. F. M. Doevendans (u. a.) | Taschenbuch | xiv | Englisch | 2010 | Springer | EAN 9789048151899 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.

Zustand: New. pp. 348 52:B&W 6.14 x 9.21in or 234 x 156mm (Royal 8vo) Case Laminate on White w/Gloss Lam.

Taschenbuch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Improving our insights into the genetic predisposition to cardiovascular disease is one of the most important challenges in our field in the next millennium, not only to unravel the cause of disease but also to improve the selection of patients for particular treatments. Nowadays, for example, subjects with a cholesterol above a particular plasma level are exposed to a cholesterol lowering regime based upon the beneficial outcome of epidemiological studies which include subjects not prone to the disease, despite a plasma cholesterol above the accepted level. Identification of the patients who are genetically predisposed to the consequences of this disorder will reduce the number of subjects unnecessarily treated and, hence, the costs of health care. Because in most cardiovascular diseases the genetic component is a consequence of more than one gene defect, only limited progress has as yet been made in identifying subjects genetically at risk. For example, in hypertension only in less than 10% of the patients the genetic defect has been identified. It has been known for quite some time that in heart and blood vessels fetal genes are as high blood pressure and upregulated or induced when they are exposed to such disorders ischemia. Little is known about the function of these genes in the cardiac and vascular adaptation to these disorders; only guesses can be made.

Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Improving our insights into the genetic predisposition to cardiovascular disease is one of the most important challenges in our field in the next millennium, not only to unravel the cause of disease but also to improve the selection of patients for particular treatments. Nowadays, for example, subjects with a cholesterol above a particular plasma level are exposed to a cholesterol lowering regime based upon the beneficial outcome of epidemiological studies which include subjects not prone to the disease, despite a plasma cholesterol above the accepted level. Identification of the patients who are genetically predisposed to the consequences of this disorder will reduce the number of subjects unnecessarily treated and, hence, the costs of health care. Because in most cardiovascular diseases the genetic component is a consequence of more than one gene defect, only limited progress has as yet been made in identifying subjects genetically at risk. For example, in hypertension only in less than 10% of the patients the genetic defect has been identified. It has been known for quite some time that in heart and blood vessels fetal genes are as high blood pressure and upregulated or induced when they are exposed to such disorders ischemia. Little is known about the function of these genes in the cardiac and vascular adaptation to these disorders; only guesses can be made.

Paperback. Zustand: Brand New. 330 pages. 9.25x6.10x0.79 inches. In Stock.

Zustand: New. The changes in gene expression lead to functional changes observed in the cardiovascular systems of animal models and humans. This title discusses changes in the level of gene expression and protein function in cardiovascular diseases. It outlines different approaches to the design of cell-type-specific gene therapy for the heart and vessels. Editor(s): Doevendans, P. A. F. M.; Reneman, Robert S.; Bilsen, Marc van. Series: Developments in Cardiovascular Medicine. Num Pages: 330 pages, biography. BIC Classification: MBGR; MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 20. Weight in Grams: 664. . 1999. Hardback. . . . . Books ship from the US and Ireland.

Zustand: New. The first invasive evaluation of cardiac arrhythmias in humans was performed in 1967 in Paris (Prof P Coumel) and Amsterdam (Prof D Durrer). In that same year, Prof Hein J J Wellens became cardiologist in the Wilhelmina Gasthuis in Amsterdam. This title highlights the scientific work of Prof Hein J J Wellens. Editor(s): Smeets, J.; Doevendans, P. A. F. M.; Josephson, M.; Kirchhof, Ch. (University Hospital Maastricht, The Netherlands); Vos, M. Num Pages: 679 pages, 107 black & white tables, biography. BIC Classification: MFG; MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly. Dimension: 254 x 203 x 38. Weight in Grams: 1645. . 2000. 2000th Edition. hardcover. . . . . Books ship from the US and Ireland.