Dirnagl ulrich (44 Ergebnisse)

Zustand: acceptable. Ausreichend/Acceptable: Exemplar mit vollständigem Text und sämtlichen Abbildungen oder Karten. Schmutztitel oder Vorsatz können fehlen. Einband bzw. Schutzumschlag weisen unter Umständen starke Gebrauchsspuren auf. / Describes a book or dust jacket that has the complete text pages (including those with maps or plates) but may lack endpapers, half-title, etc. (which must be noted). Binding, dust jacket (if any), etc may also be worn.…

Zustand: good. Befriedigend/Good: Durchschnittlich erhaltenes Buch bzw. Schutzumschlag mit Gebrauchsspuren, aber vollständigen Seiten. / Describes the average WORN book or dust jacket that has all the pages present.

Zustand: very good. Gut/Very good: Buch bzw. Schutzumschlag mit wenigen Gebrauchsspuren an Einband, Schutzumschlag oder Seiten. / Describes a book or dust jacket that does show some signs of wear on either the binding, dust jacket or pages.

Zustand: good. Befriedigend/Good: Durchschnittlich erhaltenes Buch bzw. Schutzumschlag mit Gebrauchsspuren, aber vollständigen Seiten. / Describes the average WORN book or dust jacket that has all the pages present.

Zustand: very good. Gut/Very good: Buch bzw. Schutzumschlag mit wenigen Gebrauchsspuren an Einband, Schutzumschlag oder Seiten. / Describes a book or dust jacket that does show some signs of wear on either the binding, dust jacket or pages.

2nd ed. 25.4 cm x 17.8 cm, 0 g. XII, 338 p. Hardcover. Versand aus Deutschland / We dispatch from Germany via Air Mail. Einband bestoßen, daher Mängelexemplar gestempelt, sonst sehr guter Zustand. Imperfect copy due to slightly bumped cover, apart from this in very good condition. Stamped. Neuromethods ; 120. Sprache: Englisch. …

Zustand: Sehr gut. XII, 219 Seiten. Mit 30 Figuren und 9 Tabellen. Very good copy. ISBN: 9783540403487 Sprache: Englisch Gewicht in Gramm: 990 8° (17,5-22,5 cm). Orig.-Pappband. [Hardcover / fest gebunden].

Hardcover. Zustand: Very Good. Has some light general reading/shelfwear - otherwise this is a clean, tight copy. Dispatch within 24hrs from the UK. Book.

Zustand: Very Good. 1st. Former library copy. Pages intact with possible writing/highlighting. Binding strong with minor wear. Dust jackets/supplements may not be included. Includes library markings. Stock photo provided. Product includes identifying sticker. Better World Books: Buy Books. Do Good.

Taschenbuch. Zustand: Sehr gut. 1. Alterbedingte Gebrauchsspuren !---. nein.

Hardcover. Zustand: Gut. 224 Seiten Sehr guter Zustand/ very good In englischer Sprache/ English. ha1047494 Sprache: Englisch Gewicht in Gramm: 520.

Zustand: Gut. 334 S., 8°, großformatiges Taschenbuch, Kartoniert guter Zustand: Einb. leicht bestossen. Widmung (könnte auch Signatur von Müller sein) 440 gr.

Zustand: New. In English.

Zustand: New. In English.

Zustand: New. In English.

Zustand: New. In English.

Paperback. Zustand: Brand New. reprint edition. 240 pages. 8.26x5.82x1.30 inches. In Stock.

Zustand: New. G.A. Rosenberg, Sh. Mun-Bryce: Matrix Metalloproteinases in Neuroinflammation and Cerebral Ischemia H. Wolburg et al: Involvement of Tight Junctions During Transendothelial Migration of Mononuclear Cells in Experimental Autoimmune Encephalomyelitis St. Al.

Hardcover. Zustand: Brand New. 2nd edition. 10.25x7.25x1.00 inches. In Stock.

Zustand: New. Editor(s): Dirnagl, Ulrich. Series: Neuromethods. Num Pages: 350 pages, 7 black & white illustrations, 54 colour illustrations, biography. BIC Classification: MJN; PSAN. Category: (G) General (US: Trade). Dimension: 188 x 264 x 26. Weight in Grams: 862. . 2016. 2nd ed. 2016. Hardcover. . . . . Books ship from the US and Ireland. …

Zustand: New. Helps readers understand the limitations and the opportunities of modeling stroke in rodents and enable them to conduct experiments which can not only improve our understanding of the pathophysiology of this devastating disorder but also serve as the basis for developing new highly effective treatments. Editor(s): Dirnagl, Ulrich. Series: Neuromethods. Num Pages: 286 pages, 9 black & white tables, biography. BIC Classification: MMQ. Category: (P) Professional & Vocational. Dimension: 254 x 178 x 17. Weight in Grams: 1630. . 2010. Hardback. . . . . Books ship from the US and Ireland.…

Taschenbuch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Stroke is a major cause of death and disability in industrialized countries. To date, the medical need of efficient therapy for this de vastating cerebrovascular disorder remains unmet. During the last decades, the development of pharmacological stroke therapies was aimed at improving patient outcome by restoration of cerebral blood flow or protection from acute neuronal cell death. Almost all of The participants of the workshop VI Preface these appraaches targeted the very early events after vascular occIu sion. However, primarily for logistical reasons, only a small portion of strakes can be treated within 6-10 h after the insult. In recent years it has been recognized that strake pathophysiology is a dynamic pracess, and that delayed pracesses, which occur dur ing the days and weeks following arterial occIusion, may lead to further deterioration or to impairment of recovery and rehabilitation in subacute and chranic stages. Evidence is accumulating that neu rainflammation is a major player in these delayed pathophysiologi cal pracesses. While some components of neurainflammation such as removal of cell debris and release of traphic factors may support recovery pracesses, others such as the generation of free radicals and other cytotoxic mediators are deleterious for brain tissue after isch emia. Thus, neurainflammation after strake can be considered a dou ble-edged sword, having potentially both detrimental and beneficial effects. As in other inflammatory reactions in the body, the cellular and humoral interactions are highly complex in the setting of neu rainflammation.…

Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - This second edition provides updated and expanded chapters that critically address the issues or rodent stroke modeling, from choosing the model and outcome measures, designing the experiment, conducting and analyzing it, to reporting it in a scientific publication. Rodent Models of Stroke, Second Edition aims to help its readers understand the limitations and the opportunities of modeling stroke in rodents and enable them to conduct experiments which will not only improve our understanding of the pathophysiology of this devastating disorder but also serve as the basis for developing new highly effective treatments. Written for the popular Neuromethodsseries, chapters include the kind of detail and key implementation advice that ensures successful results in the laboratory. Authoritative and practical,Rodent Models of Stroke, Second Edition provides authoritative reviews of the most commonly used, well-honed approachesin the field today.…

Zustand: New. In English.

Zustand: New. In English.

Taschenbuch. Zustand: Neu. Rodent Models of Stroke | Ulrich Dirnagl | Taschenbuch | Neuromethods | xii | Englisch | 2018 | Springer | EAN 9781493981670 | Verantwortliche Person für die EU: Humana Press in Springer Science + Business Media, Heidelberger Platz 3, 14197 Berlin, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu. …

Taschenbuch. Zustand: Neu. Optical Imaging of Brain Function and Metabolism 2 | Physiological Basis and Comparison to Other Functional Neuroimaging Methods | Arno Villringer (u. a.) | Taschenbuch | xii | Englisch | 2013 | Springer | EAN 9781489900586 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.…

Zustand: New. 2018. Paperback. . . . . . Books ship from the US and Ireland.

Zustand: Sehr gut. Zustand: Sehr gut | Sprache: Englisch | Produktart: Bücher | Stroke is a major cause of death and disability in industrialized countries. To date, the medical need of efficient therapy for this de­ vastating cerebrovascular disorder remains unmet. During the last decades, the development of pharmacological stroke therapies was aimed at improving patient outcome by restoration of cerebral blood flow or protection from acute neuronal cell death. Almost all of The participants of the workshop VI Preface these appraaches targeted the very early events after vascular occIu­ sion. However, primarily for logistical reasons, only a small portion of strakes can be treated within 6-10 h after the insult. In recent years it has been recognized that strake pathophysiology is a dynamic pracess, and that delayed pracesses, which occur dur­ ing the days and weeks following arterial occIusion, may lead to further deterioration or to impairment of recovery and rehabilitation in subacute and chranic stages. Evidence is accumulating that neu­ rainflammation is a major player in these delayed pathophysiologi­ cal pracesses. While some components of neurainflammation such as removal of cell debris and release of traphic factors may support recovery pracesses, others such as the generation of free radicals and other cytotoxic mediators are deleterious for brain tissue after isch­ emia. Thus, neurainflammation after strake can be considered a dou­ ble-edged sword, having potentially both detrimental and beneficial effects. As in other inflammatory reactions in the body, the cellular and humoral interactions are highly complex in the setting of neu­ rainflammation.…

Zustand: Sehr gut. Zustand: Sehr gut | Sprache: Englisch | Produktart: Bücher | Stroke is a major cause of death and disability in industrialized countries. To date, the medical need of efficient therapy for this de­ vastating cerebrovascular disorder remains unmet. During the last decades, the development of pharmacological stroke therapies was aimed at improving patient outcome by restoration of cerebral blood flow or protection from acute neuronal cell death. Almost all of The participants of the workshop VI Preface these appraaches targeted the very early events after vascular occIu­ sion. However, primarily for logistical reasons, only a small portion of strakes can be treated within 6-10 h after the insult. In recent years it has been recognized that strake pathophysiology is a dynamic pracess, and that delayed pracesses, which occur dur­ ing the days and weeks following arterial occIusion, may lead to further deterioration or to impairment of recovery and rehabilitation in subacute and chranic stages. Evidence is accumulating that neu­ rainflammation is a major player in these delayed pathophysiologi­ cal pracesses. While some components of neurainflammation such as removal of cell debris and release of traphic factors may support recovery pracesses, others such as the generation of free radicals and other cytotoxic mediators are deleterious for brain tissue after isch­ emia. Thus, neurainflammation after strake can be considered a dou­ ble-edged sword, having potentially both detrimental and beneficial effects. As in other inflammatory reactions in the body, the cellular and humoral interactions are highly complex in the setting of neu­ rainflammation.…