De broe m e (19 Ergebnisse)

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

Zustand: New. In English.

Zustand: NEW.

Zustand: New. In English.

Taschenbuch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Prevention of renal disease: an overview.- Evaluation and significance of renal functional reserve in health and disease.- Angiotensin I: Converting enzyme inhibition in models of progressive glomerulosclerosis.- Prevention of nephrotoxicity of beta-lactam antibiotics.- Prevention of analgesic nephropathy.- Efficacy and nephrotoxicity of aminoglycoside.- Prevention of cadmium and lead nephropathy.- Prevention of bone disease in renal failure.- Kidney function, hypertension and clinical diabetic nephropathy in type I diabetes.- Drug management of hyperlipidemia.- Index of subjects. …

Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Prevention of renal disease: an overview.- Evaluation and significance of renal functional reserve in health and disease.- Angiotensin I: Converting enzyme inhibition in models of progressive glomerulosclerosis.- Prevention of nephrotoxicity of beta-lactam antibiotics.- Prevention of analgesic nephropathy.- Efficacy and nephrotoxicity of aminoglycoside.- Prevention of cadmium and lead nephropathy.- Prevention of bone disease in renal failure.- Kidney function, hypertension and clinical diabetic nephropathy in type I diabetes.- Drug management of hyperlipidemia.- Index of subjects.…

Zustand: New. Prevention of renal disease: an overview.- Evaluation and significance of renal functional reserve in health and disease.- Angiotensin I: Converting enzyme inhibition in models of progressive glomerulosclerosis.- Prevention of nephrotoxicity of beta-lactam .

Zustand: New. Editor(s): De Broe, M. E.; Verpooten, Gert A. Series: Developments in Nephrology. Num Pages: 112 pages, biography. BIC Classification: MJR. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly. Dimension: 234 x 156 x 7. Weight in Grams: 354. . 1991. Hardback. . . . . Books ship from the US and Ireland. …

Zustand: New.

Paperback. Zustand: Brand New. 3rd reprint edition. 987 pages. 10.98x8.27x1.98 inches. In Stock.

Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Clinical Nephrotoxins: Renal Injury from Drugs and Chemicals, Third Edition is a comprehensive text on all aspects of adverse effects by drugs, chemical substances and radiation on the kidneys. The importance of the toxicity of drugs and other substances for the kidneys is increasingly recognized. The book consists of themes such as clinical relevance, renal handling and cellular mechanisms of nephrotoxicity, as well as animal and cell culture models. In addition, the volume highlights specific types of drugs, such as anti-infectious agents and anti-inflammatory, cardiovascular and anti-cancer drugs. The last section deals with prevention and focuses on urinary biomarkers, pharmacological aspects and drug dosage in renal failure. In this third and completely revised edition, several new topics were added and additional figures and tables will be included.The book is of interest for the nephrologist, internist, general practitioner, toxicologist, pharmacologist, anesthesiologist, epidemiologist, public health official, pharmaceutical industry, and national drug safety committees, among others.…

Zustand: New. In English.

Zustand: New. In English.

Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Increasing the accumulation of aluminum in the bone (body) in cases of renal osteodystrophy may influence the histopathologic aspect of the bones. Alumi num blunts the effect of increased PTH secretion and favours the genesis of osteoid. That means, in cases of renal failure combined with aluminum accumulation, a relatively low bone tunover is found and no fibrosis of the bone marrow. Furthermore the amount of osteoid is increased. This means that there is evidence of osteomalacia especially when the latter is defined as an increased amount of osteoid covered with a relatively low number of cubic osteoblasts. To a certain extent the effect of aluminum accumulation is comparable to the effect of PTX. Treatment with DFO may normalize the bone, although not necessarily with a concomittant disappearance of alumi num from the bone. The presence of aluminum in the bone can be suggested by routine histologic investigation of the bone and can be made rather probably by the aluminum staining combined with iron-staining, but can only be proven by more advanced techniques like ET AAS and LAMMA. References 1. Boyce BF, Elder HY, Elliot HL, Fogelman I, Gell GS, lunor Bl, Beastall G, Boyle YT, 1982: Hypercaicaemic ostemalacia due to aluminium toxicity. Lancet 6: 1009. 2. Verbueken AH, Visser Wl, Van de Vyver FL, Van Grieken RE, De Broe ME, 1986: The use of laser microprobe mass analysis (LAMMA) to control the staining of aluminum by aurin tricarboxylate (aluminon). Stain Technology 61: 287.…

Zustand: New. General.- Historical survey of aluminum-related diseases.- Chemistry of aluminum.- The metabolism of aluminum.- Intestinal absorption of aluminum.- Aluminum determination in biological samples.- Quality control of aluminum measurements.- Pathophysiology.- P.

Paperback. Zustand: Brand New. 392 pages. 9.25x6.10x0.93 inches. In Stock.

Hardcover. Zustand: Brand New. 388 pages. 9.13x6.57x1.11 inches. In Stock.

Taschenbuch. Zustand: Neu. Neuware - Increasing the accumulation of aluminum in the bone (body) in cases of renal osteodystrophy may influence the histopathologic aspect of the bones. Alumi num blunts the effect of increased PTH secretion and favours the genesis of osteoid. That means, in cases of renal failure combined with aluminum accumulation, a relatively low bone tunover is found and no fibrosis of the bone marrow. Furthermore the amount of osteoid is increased. This means that there is evidence of osteomalacia especially when the latter is defined as an increased amount of osteoid covered with a relatively low number of cubic osteoblasts. To a certain extent the effect of aluminum accumulation is comparable to the effect of PTX. Treatment with DFO may normalize the bone, although not necessarily with a concomittant disappearance of alumi num from the bone. The presence of aluminum in the bone can be suggested by routine histologic investigation of the bone and can be made rather probably by the aluminum staining combined with iron-staining, but can only be proven by more advanced techniques like ET AAS and LAMMA. References 1. Boyce BF, Elder HY, Elliot HL, Fogelman I, Gell GS, lunor Bl, Beastall G, Boyle YT, 1982: Hypercaicaemic ostemalacia due to aluminium toxicity. Lancet 6: 1009. 2. Verbueken AH, Visser Wl, Van de Vyver FL, Van Grieken RE, De Broe ME, 1986: The use of laser microprobe mass analysis (LAMMA) to control the staining of aluminum by aurin tricarboxylate (aluminon). Stain Technology 61: 287.…

Hardcover. Zustand: Good. pp. 1075 Dutch.