9783764358754 - pain and neurogenic inflammation (progress in inflammation research) (4 Ergebnisse)
Sprache: Englisch
Verlag: Birkhäuser, 1998
Serie: Progress in Inflammation Research, Buch 32 von 50. Buch 32 von 50 - Progress in Inflammation Research
- Hardcover
Anbieter: Ria Christie Collections, Uxbridge, Vereinigtes KönigreichRia Christie Collections
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Zustand: New. In.
Sprache: Englisch
Verlag: Birkhäuser Basel, 1998
Serie: Progress in Inflammation Research, Buch 32 von 50. Buch 32 von 50 - Progress in Inflammation Research
- Hardcover
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Zustand: New. A comparative discussion of A? and C fibres in different tissues.- The roles of spinal receptors in nociceptive responses.- Cutaneous hyperalgesia.- Capsaicin and pain mechanisms.- Nitric oxide and inflammatory pain.- Interactions between kinins and the inf.
Sprache: Englisch
Verlag: Birkhauser Verlag AG, 1998
Serie: Progress in Inflammation Research, Buch 32 von 50. Buch 32 von 50 - Progress in Inflammation Research
- Hardcover
Anbieter: Kennys Bookstore, Olney, MD, USAKennys Bookstore
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Zustand: New. Aims to bring together advances in the often separate fields of pain and neurogenic inflammation. This work focuses on important discoveries such as the cloning of the capsaicin receptor and the discovery of RAMP proteins for CGRP receptors. It provides an integrated account of advances in the fields of pain and ne…urogenic inflammation. Editor(s): Moore, Phillip K. Series: Progress in Inflammation Research. Num Pages: 360 pages, biography. BIC Classification: MMBP; MMG. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 234 x 156 x 20. Weight in Grams: 1510. . 1998. Hardback. . . . . Books ship from the US and Ireland.
Sprache: Englisch
Verlag: Springer, Basel, Birkhäuser Basel, Birkhäuser, 1998
Serie: Progress in Inflammation Research, Buch 32 von 50. Buch 32 von 50 - Progress in Inflammation Research
- Hardcover
Anbieter: AHA-BUCH GmbH, Einbeck, DeutschlandAHA-BUCH GmbH
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Buch. Zustand: Neu. Neuware - Pain and inflammation are inextricably linked phenomena. The observation that chemical mediators with combined pro-inflammatory, algesic and/or hyperalgesic activity occur at the site of inflammation is fundamental not only to our present understanding of the inflammatory process but also to our att…empts to devise clini cally useful anti-inflammatory therapies. Over a hundred years ago it was recognised that primary sensory neurones play a crucially important 'dual' role in inflammation. By affecting the transfer of infor mation from peripheral nociceptors to the spinal cord, a subpopulation of sensory nerves {'pain fibres'} initiate algesia and hyperalgesia, whose sensations are then modified and fine-tuned in the central nervous system. Equally important is the release from the peripheral terminals of sensory neurones of neuropeptides, the acute effects of which are observed as changes in microvascular tone and perme ability leading to neurogenic inflammation. Over the last decade it has become increasingly clear that this view of the func tion of sensory nerves is somewhat over-simplified. For example, the mechanisms underlying hyperalgesia may, in certain circumstances, be mimicked in other condi tions such as the hypersensivity associated with asthma. Furthermore, it has become increasingly evident that over a longer time period the release of neuropeptides from peripheral sensory nerve endings may also have modulating effects on inmune cells and that this may be relevant to chronic inflammatory disease and possibly also to inflammatory hyperalgesia.
