Anbieter: Ria Christie Collections, Uxbridge, Vereinigtes Königreich
EUR 115,14
Anzahl: Mehr als 20 verfügbar
In den WarenkorbZustand: New. In.
Anbieter: Revaluation Books, Exeter, Vereinigtes Königreich
EUR 153,30
Anzahl: 2 verfügbar
In den WarenkorbHardcover. Zustand: Brand New. 248 pages. 9.10x6.50x0.80 inches. In Stock.
Anbieter: Kennys Bookstore, Olney, MD, USA
Zustand: New. The goal of this text is to outline the major arguments and data suggesting that T cells may, or may not, be central players in the pathogensis of chronic rheumatoid arthritis. Editor(s): Miossec, P.; Berg, W. van den. Series: Progress in Inflammation Research. Num Pages: 248 pages, biography. BIC Classification: MFC; MNS. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 16. Weight in Grams: 1050. . 1998. Hardback. . . . . Books ship from the US and Ireland.
Sprache: Englisch
Verlag: Birkhäuser Basel, Springer Basel, 1998
ISBN 10: 376435853X ISBN 13: 9783764358532
Anbieter: AHA-BUCH GmbH, Einbeck, Deutschland
Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Rheumatoid arthritis (RA) is the most common and most severe form of inflammatory arthritis. The pathogenesis of RA has been the subject of intense research for several decades. The prevailing hypotheses have changed over the years, and have attempted to incorporate the most recent data. Although T cells represent an important component of the cells which infiltrate the joint synovium, their contribution at a late stage of the disease remains a matter of debate. The goal of this book is to outline the major arguments and data suggesting that T cells may, or may not, be central players in the pathogenesis of chronic RA. While each of the editors and authors has his/her own bias (as will be clear by reading the respective chapters), our hope is that the readers will enjoy a complete and balanced view of the critical questions and experiments. This is not just an intellectual exercise since the direction of future therapeutic interventions depends heavily on how one interprets the pathogenesis of RA and the contribution of T cells.