9781402012235 - the role of aging in atherosclerosis: the sequestration hypothesis von tracy, r.e. (3 Ergebnisse)

- Hardcover
Anbieter: Ria Christie Collections, Uxbridge, Vereinigtes KönigreichRia Christie Collections
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EUR 116,63
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Zustand: New. In.

- Hardcover
Anbieter: Kennys Bookstore, Olney, MD, USAKennys Bookstore
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Zustand: New. Addressing aging as a risk factor, this book provides observations and insights. It deals with topics such as the biology of aging, atherosclerosis, hypertension, and diabetes, including the microvascular features of these disorders. It is useful for those studying these topics. Num Pages: 273 pages, biography. BIC… Classification: MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 17. Weight in Grams: 581. . 2003. Hardback. . . . . Books ship from the US and Ireland.

- Hardcover
Anbieter: AHA-BUCH GmbH, Einbeck, DeutschlandAHA-BUCH GmbH
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EUR 114,36
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Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - The cover of this book summarizes the central features of the sequestration hypothesis: Commonplace appearances seen in human coronary artery, fat stained in paraffin seetions by a new technique explained in Chapter Eleven, are arranged to suggest pathways…of evolution toward atheroma. The hypothesis formulated and defended in the pages ofthis book is this: Fibroplasia progresses upward in column 'a' from 'la' to '3a' as a characteristic feature of aging. This starts sooner and progresses faster in men than in wornen. Numbers ofSMC's remain essentially constant so that fibroplasia per SMC steadily increases. The rise upward conveys an increasing propensity to sequester atherogenic lipids, causing transition rightward into column 'b'. Sequestered extracellular lipid then attracts fatty streak elements, especially foam cells and lyrnphocytes, to propel the arterial site rightward into column 'c'. Frame 'lc' corresponds to the AHA Lesions Committee classification type IIb, the progression resistant fatty streak arising directly without prior lipid sequestration; this can progress to atheroma, but slowly after much delay, although extreme provocation can accelerate the process. Such progression is rightward toward atherorna with thin cap, not upward toward fibroplastic thickening. Frame '2c' corresponds to the AHA classification, type Ha, progression prone fatty streaks. These readily evolve into atheroma, again by horizontal progression.