EUR 40,76
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In den WarenkorbZustand: New. pp. 524 102 Illus. (43 Col.).
Zustand: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.
Zustand: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.
Taschenbuch. Zustand: Neu. Molecular Mechanisms of Synaptogenesis | Alaa El-Husseini (u. a.) | Taschenbuch | xxvi | Englisch | 2010 | Springer US | EAN 9781441940902 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
Anbieter: Majestic Books, Hounslow, Vereinigtes Königreich
EUR 227,33
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In den WarenkorbZustand: New. pp. 508.
Verlag: Springer US, Springer New York, 2010
ISBN 10: 1441940901 ISBN 13: 9781441940902
Sprache: Englisch
Anbieter: AHA-BUCH GmbH, Einbeck, Deutschland
Taschenbuch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Some of the molecules critical for building neuronal contacts have been shown to be affected in psychiatric disorders such as autism and mental retardation. Also, a reduction in synapse number in specific brain regions has been found in patients with brain disorders. Thus, imbalance in synaptic contact formation may result in the formation of abnormal neuronal circuits and the aberrant behavior manifested in these brain diseases. Recent findings also suggest that fine tuning of neuronal circuits rather than a major loss of neurons may be the cause of some of these brain disorders. In certain cases, loss of neurons could be a secondary event reflecting problems in improper neuronal communication.
Anbieter: Revaluation Books, Exeter, Vereinigtes Königreich
EUR 259,80
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In den WarenkorbHardcover. Zustand: Brand New. 1st edition. 472 pages. 9.50x7.75x1.25 inches. In Stock.
Buch. Zustand: Neu. Neuware - Some of the molecules critical for building neuronal contacts have been shown to be affected in psychiatric disorders such as autism and mental retardation. Also, a reduction in synapse number in specific brain regions has been found in patients with brain disorders. Thus, imbalance in synaptic contact formation may result in the formation of abnormal neuronal circuits and the aberrant behavior manifested in these brain diseases. Recent findings also suggest that fine tuning of neuronal circuits rather than a major loss of neurons may be the cause of some of these brain disorders. In certain cases, loss of neurons could be a secondary event reflecting problems in improper neuronal communication.