Helicobacter pylori: Basic Mechanisms to Clinical Cure 2000 - Softcover

Inhaltsangabe

Section I: Helicobacter pylori - The Organism.- 1 What are the biochemical and physiological implications of the new genetic information?.- 2 The urease system of Helicobacter pylori.- 3 The amphibiotic relationship of Helicobacter pylori and humans.- 4 Helicobacter pylori is pathogenic flora.- 5 Disease-specific Helicobacter pylori virulence factors: the role of cagA, vacA, iceA, babA2 alone or in combination.- Section II: Helicobacter pylori - Epidemiology.- 6 Factors associated with disappearance of Helicobacter pylori in the West.- 7 Factors associated with disappearance of Helicobacter pylori in the Far East.- 8 Differences in prevalence of Helicobacter pylori and disease outcomes according to race/environmental factors in Southeast Asia.- Section III: Novel Helicobacters.- 9 Infection with Helicobacter heilmannii (formerly Gastrospirillum hominis): characterization, epidemiology and therapy.- 10 Hepatobiliary Helicobacters: recognized animal pathogens with suspected pathogenic potential in humans.- 11 Novel Helicobacter species in the intestine.- Section IV: Diagnosis of Helicobacter pylori Infection.- 12 Diagnosis of Helicobacter pylori infection: faecal antigen determination.- 13 Pitfalls in Helicobacter pylori diagnosis.- Section V: Inflammation and the Immune Response to Helicobacter pylori Infection.- 14 Overview of immune and inflammatory changes due to Helicobacter infection.- 15 Interaction of Helicobacter pylori with gastric epithelium.- 16 Helicobacter pylori and the epithelial barrier: role of oxidative injury.- 17 Immuno-inflammatory response to Helicobacter pylori in children.- 18 Severity and reversibility of mucosal inflammation in children and adolescents infected with Helicobacter pylori.- 19 Elimination of Helicobacter pylori is dependent on a Th2 response.- 20 Elimination of Helicobacter pylori is not dependent on a Th2 cytokine response.- 21 The inflammatory activity in Helicobacter pylori infection is predominantly organism related.- 22 The inflammatory activity in Helicobacter pylori infection is predominantly host-related.- Section VI: Helicobacter pylori and Gastritis.- 23 Helicobacter pylori gastritis - a global view.- 24 Unusual forms of gastric inflammation and their relationship to Helicobacter pylori infection.- 25 Can atrophic gastritis be diagnosed in the presence of Helicobacter pylori infection?.- 26 Mechanisms involved in gastric atrophy.- 27 Intestinal metaplasia: types, mechanisms of origin, and role in gastric cancer histogenesis.- 28 Long-term proton pump inhibitor therapy accelerates the onset of atrophic gastritis in Helicobacter pylori-positive patients.- 29 Proton pump inhibitors do not accelerate the development of gastric atrophy in Helicobacter pylori gastritis.- 30 Autoimmune gastritis via mimicking does occur.- 31 Autoimmune gastritis and antigenic mimicking.- 32 Carditis and intestinal metaplasia of the cardia is reflux related.- 33 Carditis and cardia intestinal metaplasia are Helicobacter pylori-related.- 34 Is gastric metaplasia in Helicobacter pylori really gastric?.- Section VII: Helicobacter pylori and Clinical Consequences.- 35 Extragastric manifestations of Helicobacter pylori - are they relevant?.- 36 Peptic ulcer disease - the transitional zones are important.- 37 What causes Helicobacter pylori-negative non-NSAID-related ulcers?.- 38 From the pump to the helix.- 39 Mechanisms involved in the development of hypochlorhydria and pangastritis in Helicobacter pylori infection.- 40 Effect of Helicobacter pylori infection on gastric acid control using proton pump inhibitors.- 41 Rebound acid hypersecretion after acid-suppressive therapy.- 42 Gastric consequences of proton pump therapy and Helicobacter pylori eradication.- Section VIII: Helicobacter pylori, Dyspepsia and NSAIDs.- 43 Current concepts of dyspepsia: the role of the nervous system.- 44 How to explain outcome differences in dyspepsia studies.- 45 Helicobacter pylori eradication for dyspepsia is clinically use

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