Homonymous Visual Field Defects - Hardcover

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Karolína Skorkovská, MD, PhD, graduated from the Faculty of Medicine, Masaryk University, Brno, Czech Republic, in 2002. She then undertook postgraduate studies in the Department of Ophthalmology and Optometry, Masaryk University and St. Ann University Hospital, Brno, completing a thesis on the importance of structural examination methods in the follow-up of patients with ocular hypertension. In 2009 Dr. Skorkovská passed the Specialization Examination in Ophthalmology at the Institute of Postgraduate Education, Prague. She continues to work at the Department of Ophthalmology and Optometry, Masaryk University/St. Ann University Hospital, Brno, specializing in glaucoma, neuro-ophthalmology, and cataract surgery. She has been the recipient of a Marie Curie Fellowship and of a Fellowship from the Jung Foundation. Dr. Skorkovská has published almost 20 articles in national and international journals and received an award from the Czech Glaucoma Society for the best publication on glaucomain 2008.

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This book provides a concise and up-to-date overview on homonymous visual field defects, with the focus especially on homonymous hemianopia. All aspects of the subject are covered, from anatomy and pathophysiology to diagnosis and management. Emphasis is placed on practical issues and major advances in ophthalmology, neurology, imaging techniques, and psychology. Rehabilitation strategies are described, and methods supporting compensation strategies are recommended. A separate chapter considers driving with homonymous hemianopia, which represents one of the critical issues in traffic ophthalmology.

Homonymous Visual Field Defects will be of particular value to ophthalmologists, neurologists, neurosurgeons, and neuroscientists. It will be an invaluable asset in diagnostic and treatment decision making in patients with such defects, which occur primarily as a result of stroke, trauma, or tumor, cause reading and orientation disorders, and are the most frequent type of visual field deficit after acquired unilateral postchiasmal brain damage.